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American Heart Journal
Volume 144, Issue 6
, Pages 938-940
, December 2002
Genetics in heart failure: Practical incorporation of this new biologic dimension
References
- A novel lamin A/C mutation in a family with dilated cardiomyopathy, prominent conduction system disease, and need for permanent pacemaker implantation. Am Heart J. 2002;144:1081–1086
- Missense mutations in the rod domain of the lamin A/C gene as causes of dilated cardiomyopathy and conduction system disease. N Engl J Med. 1999;341:1715–1724
- Prognostic impact of diabetes mellitus in patients with heart failure according to the etiology of the left ventricular systolic dysfunction. J Am Coll Cardiol. 2001;38:421–428
-
Mitral regurgitation as an independent risk factor for mortality in patients with heart failure and left ventricular systolic dysfunction [abstract].
J Am Coll Cardiol. 2002;39(A Suppl):194A
- The Ile164 beta-2 adrenergic polymorphism adversely affects the outcome of congestive heart failure. J Clin Invest. 1998;102:1534–1539
- Polymorphisms of the beta(2)-adrenergic receptor determine exercise capacity in patients with heart failure. Circ Res. 2000;86:834–840
- Common variant in AMPD1 gene predicts improved clinical outcome in patients with heart failure. Circulation. 1999;99:1422–1425
- A polymorphism in the endothelin-A receptor gene predicts survival in patients with idiopathic dilated cardiomyopathy. Eur Heart J. 2001;22:1948–1953
☆ Reprint requests: Reprints not available from authors.
PII: S0002-8703(02)00217-X
doi: 10.1067/mhj.2002.126738
© 2002 Mosby, Inc. All rights reserved.
« Previous
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American Heart Journal
Volume 144, Issue 6
, Pages 938-940
, December 2002
